107 Genetic Conditions,
Biologically Validated

Every condition is simulated with reference-backed hg38 sequences, aligned with BWA, and validated through category-specific detection pipelines at 95%+ alignment rate. Validated for synthetic data generation — not yet validated in clinical patient samples.

107Conditions Modelled
91%E2E Detection Rate
95%+Alignment Rate
7Condition Categories

14 Aneuploidy Conditions

Whole chromosome copy number changes detectable via coverage-based z-scores. Includes common trisomies, sex chromosome aneuploidies, rare autosomal trisomies, and complex karyotypes.

100%Detected
Detection methodChromosome z-score
ConditionGene / RegionMetricPrevalenceNotes
Trisomy 21 OMIMchr21z = 7.661 in 700Most common viable autosomal trisomy
Trisomy 18 OMIMchr18z = 5.531 in 5,000Edwards syndrome
Trisomy 13 OMIMchr13z = 7.481 in 16,000Patau syndrome
45,X (Turner syndrome) OMIMchrXz = -6.461 in 2,500Monosomy X
47,XXY (Klinefelter) OMIMchrXz = 2.681 in 660Sex chromosome aneuploidy
47,XXX (Triple X) OMIMchrXz = 5.891 in 1,000Sex chromosome aneuploidy
47,XYY OMIMchrYz = 9.471 in 1,000Sex chromosome aneuploidy
Trisomy 8 OMIMchr8z = 3.22RareOften mosaic
Trisomy 9 OMIMchr9z = 8.36RareRare autosomal trisomy
Trisomy 10chr10z = 5.36Very rareRare autosomal trisomy
Trisomy 16chr16z = 6.98RareMost common trisomy in miscarriage
Trisomy 22 OMIMchr22z = 4.12RareRare autosomal trisomy
Triploidy (69,XXX/XXY)genome-widez = multi-chr1 in 10,000Complete extra haploid set
48,XXYY syndromechrX/chrYz = 2.691 in 18,000Complex sex chromosome aneuploidy

24 Microdeletion Conditions

Sub-chromosomal deletions detected via regional log2 coverage ratios with Poisson statistical testing. GC-normalised for accurate signal quantification across deletions from 450 kb to 21 Mb.

100%Detected
Detection methodRegional log2 ratio + Poisson test
ConditionGene / RegionMetricPrevalenceNotes
DiGeorge syndrome (3.0 Mb A-D) OMIM22q11.2log2 = -0.111 in 4,0003.0 Mb deletion, ~85-90% of cases. LCR22A-D breakpoints. GC-rich region with mappability-aware modelling.
DiGeorge syndrome (1.5 Mb A-B) OMIM22q11.2log2 = -0.071 in 4,0001.5 Mb deletion, ~5-8% of cases. LCR22A-B breakpoints.
DiGeorge syndrome (2.0 Mb A-C) OMIM22q11.2log2 = -0.091 in 4,0002.0 Mb deletion, ~2-3% of cases. LCR22A-C breakpoints.
Cri du Chat OMIM5plog2 = -0.101 in 50,000Terminal 5p deletion
Williams syndrome OMIM7q11.23log2 = -0.071 in 10,0001.4 Mb ELN region
Prader-Willi syndrome OMIM15q11.2log2 = -0.091 in 15,000Paternal deletion
Angelman syndrome OMIM15q11.2log2 = -0.091 in 15,000Maternal deletion
Wolf-Hirschhorn OMIM4p16.3log2 = -0.141 in 50,000Terminal 4p deletion
Smith-Magenis OMIM17p11.2log2 = -0.111 in 25,000RAI1 region
Jacobsen syndrome OMIM11q24log2 = -0.121 in 100,000Terminal 11q deletion, 21 Mb
Langer-Giedion OMIM8q24log2 = -0.15RareTRPS1/EXT1 region
1p36 deletion OMIM1p36log2 = -0.111 in 5,000Most common terminal deletion
16p11.2 deletion OMIM16p11.2log2 = -0.431 in 2,000Autism-associated, 600 kb
2q37 deletion OMIM2q37log2 = -0.11RareBrachydactyly-mental retardation
3q29 deletion OMIM3q29log2 = -0.241 in 30,000Psychiatric phenotype
Miller-Dieker syndrome OMIM17p13.3log2 = -0.121 in 100,000LIS1/YWHAE, lissencephaly
Koolen-de Vries syndrome OMIM17q21.31log2 = -0.101 in 55,000KANSL1, 624 kb
15q11.2 BP1-BP2 (Burnside-Butler) OMIM15q11.2log2 = -0.081 in 200NIPA1, 452 kb, incomplete penetrance
15q13.3 deletion OMIM15q13.3log2 = -0.101 in 40,000CHRNA7, epilepsy risk
16p12.2 deletion OMIM16p12.2log2 = -0.09RareCDR2/EEF2K, 500 kb
Rubinstein-Taybi syndrome OMIM16p13.3log2 = -0.111 in 125,000CREBBP, 3.75 Mb
Glass syndrome OMIM2q33.1log2 = -0.13RareSATB2, 8.1 Mb
NRXN1 deletion OMIM2p16.3log2 = -0.10RareAutism/schizophrenia risk, 1.1 Mb
X-linked ichthyosis OMIMXp22.31log2 = -0.111 in 6,000 malesSTS deletion, 1.7 Mb

4 Microduplication Conditions

Sub-chromosomal duplications detected via positive regional log2 coverage ratios. Inverse of microdeletion detection with increased coverage in the duplicated region.

100%Detected
Detection methodRegional log2 ratio (positive)
ConditionGene / RegionMetricPrevalenceNotes
22q11.2 duplication OMIM22q11.2log2 = +0.101 in 7002.6 Mb, variable phenotype
7q11.23 duplication OMIM7q11.23log2 = +0.08RareReciprocal of Williams deletion
Potocki-Lupski syndrome OMIM17p11.2log2 = +0.101 in 25,000Reciprocal of Smith-Magenis
16p11.2 duplication OMIM16p11.2log2 = +0.091 in 2,000Reciprocal of 16p11.2 deletion

49 Monogenic Conditions

Single-gene disorders detected via variant allele frequency (VAF) in cell-free DNA. Includes SNVs, small indels, and structural deletions. Covers haemoglobinopathies, skeletal dysplasias, RASopathies, connective tissue disorders, neurodevelopmental conditions, and treatable metabolic diseases.

100%Detected
Detection methodVariant allele frequency (VAF)
ConditionGene / RegionMetricPrevalenceNotes
Sickle Cell Anemia OMIMHBBVAF = 0.0701 in 500 (AFR)E6V missense
Cystic Fibrosis OMIMCFTRVAF = 0.0551 in 2,500 (EUR)F508del
Beta-thalassemia OMIMHBBVAF = 0.0951 in 100,000Splice site variant
Alpha-thalassemia OMIMHBA1/HBA2log2 = -0.241 in 10,000SEA deletion (structural)
Spinal Muscular Atrophy OMIMSMN1log2 = -0.151 in 10,000Exon 7 deletion (structural)
Tay-Sachs Disease OMIMHEXAVAF = 0.1001 in 3,500 (AJ)1278insTATC
Achondroplasia OMIMFGFR3VAF = 0.0701 in 15,000G380R de novo
Pfeiffer syndrome OMIMFGFR1VAF = 0.0651 in 100,000R661X de novo
Thanatophoric dysplasia I OMIMFGFR3VAF = 0.0701 in 60,000R248C / S249C de novo
Hypochondroplasia OMIMFGFR3VAF = 0.0681 in 15,000N540K missense
Osteogenesis imperfecta (COL1A1) OMIMCOL1A1VAF = 0.0721 in 15,000Splice variant, AD
Osteogenesis imperfecta (COL1A2) OMIMCOL1A2VAF = 0.0701 in 15,000Splice variant, AD
Stickler syndrome OMIMCOL2A1VAF = 0.0681 in 7,500G504S missense
Noonan syndrome (PTPN11) OMIMPTPN11VAF = 0.0751 in 2,500N308D missense
Noonan syndrome (RAF1) OMIMRAF1VAF = 0.070RareS257L, cardiac phenotype
Noonan syndrome (KRAS) OMIMKRASVAF = 0.065RareG12C de novo
Costello syndrome OMIMHRASVAF = 0.0721 in 300,000G13D de novo, RASopathy
CFC syndrome OMIMBRAFVAF = 0.0681 in 810,000Q257R de novo, RASopathy
Marfan syndrome OMIMFBN1VAF = 0.0651 in 5,000C1039Y missense
Neurofibromatosis type 1 OMIMNF1VAF = 0.0701 in 3,000R1947X nonsense
Rett syndrome OMIMMECP2VAF = 0.0851 in 10,000R168X, X-linked de novo
Dravet syndrome OMIMSCN1AVAF = 0.0701 in 15,000F1846L de novo
DYRK1A-related ID OMIMDYRK1AVAF = 0.068RareR255X de novo
Coffin-Siris syndrome OMIMARID1BVAF = 0.072RareR1990X de novo
KAT6A syndrome OMIMKAT6AVAF = 0.070RareR1129X de novo
Duchenne Muscular Dystrophy OMIMDMDlog2 = -0.281 in 3,500 malesExon 45 deletion (structural)
Hemophilia A OMIMF8VAF = 0.0801 in 5,000 malesIntron 22 inversion
Tuberous sclerosis OMIMTSC2VAF = 0.0901 in 6,000R1743Q missense
Cornelia de Lange OMIMNIPBLVAF = 0.0651 in 10,000R2298X de novo
Treacher Collins OMIMTCOF1VAF = 0.0751 in 50,000K1060X nonsense
CHARGE syndrome OMIMCHD7VAF = 0.0801 in 10,000R2178X de novo
Smith-Lemli-Opitz OMIMDHCR7VAF = 0.0601 in 20,000Splice site, AR
Phenylketonuria OMIMPAHVAF = 0.0701 in 10,000R408W missense, treatable
Gaucher Disease OMIMGBAVAF = 0.0951 in 40,000N370S, Ashkenazi founder
Canavan Disease OMIMASPAVAF = 0.0751 in 6,400 (AJ)E285A missense
Bloom syndrome OMIMBLMVAF = 1.0001 in 48,000 (AJ)blmAsh deletion
Niemann-Pick type C OMIMNPC1VAF = 0.0801 in 120,000I1061T missense
Fanconi Anemia OMIMFANCAVAF = 0.0551 in 160,000E1439K splice variant
Mucopolysaccharidosis I OMIMIDUAVAF = 0.0951 in 100,000W402X nonsense
Alport syndrome OMIMCOL4A5VAF = 1.0001 in 50,000G325R, X-linked
Polycystic Kidney Disease OMIMPKHD1VAF = 0.0401 in 20,000T36M missense, ARPKD
Pompe Disease OMIMGAAVAF = 0.0651 in 40,000E176RfsX45, treatable (ERT)
Adrenoleukodystrophy OMIMABCD1VAF = 0.0701 in 17,000Q472RfsX83, X-linked, treatable
Galactosaemia OMIMGALTVAF = 0.0751 in 60,000Q188R, treatable (diet)
Maple Syrup Urine Disease OMIMBCKDHAVAF = 0.0681 in 185,000Y438N, treatable (diet)
MCADD OMIMACADMVAF = 0.0721 in 15,000K329E, treatable (diet)
Biotinidase Deficiency OMIMBTDVAF = 0.0701 in 60,000R538C, treatable (biotin)
Congenital Adrenal Hyperplasia OMIMCYP21A2VAF = 0.0651 in 15,000Q319X, treatable (cortisol)
Alagille Syndrome OMIMJAG1VAF = 0.0681 in 30,000C1085Y, AD

10 Oncology Hotspots Conditions

Somatic driver mutations for liquid biopsy validation. VAF represents tumour fraction rather than foetal fraction. Covers the most clinically actionable mutations across NSCLC, CRC, melanoma, breast, and pan-cancer.

100%Detected
Detection methodVariant allele frequency (VAF)
ConditionGene / RegionMetricPrevalenceNotes
EGFR L858R OMIMEGFRVAF = 0.070Common in NSCLCLung cancer driver, TKI-sensitive
EGFR T790M OMIMEGFRVAF = 0.065NSCLC resistanceThird-gen TKI target
KRAS G12D OMIMKRASVAF = 0.072CRC/PancreaticMost common KRAS mutation
KRAS G12V OMIMKRASVAF = 0.070CRC/PancreaticSecond most common KRAS
BRAF V600E OMIMBRAFVAF = 0.075Melanoma/CRCBRAF inhibitor target
PIK3CA H1047R OMIMPIK3CAVAF = 0.068Breast cancerKinase domain hotspot
PIK3CA E545K OMIMPIK3CAVAF = 0.070Breast cancerHelical domain hotspot
TP53 R273C OMIMTP53VAF = 0.072Pan-cancerContact mutant, GOF
TP53 R248W OMIMTP53VAF = 0.068Pan-cancerStructural mutant, GOF
NRAS Q61R OMIMNRASVAF = 0.070Melanoma/AMLRAS pathway oncogene

4 Repeat Expansion Conditions

Trinucleotide repeat expansions detected via repeat motif counting in aligned reads and soft-clipped bases. Targeted fragment generation ensures coverage at the repeat locus.

100%Detected
Detection methodRepeat motif detection
ConditionGene / RegionMetricPrevalenceNotes
Fragile X syndrome OMIMFMR1190 CGG repeats1 in 4,000 malesCGG expansion (>200 repeats)
Huntington Disease OMIMHTT194 CAG repeats1 in 10,000CAG expansion (>36 repeats)
Myotonic Dystrophy Type 1 OMIMDMPK80 CTG repeats1 in 8,000CTG expansion (>50 repeats)
Spinocerebellar Ataxia Type 1 OMIMATXN155 CAG repeats1 in 100,000CAG expansion (>39 repeats)

2 Imprinting Conditions

Imprinting disorders detected via regional coverage changes and fragment size distribution shifts at imprinted loci.

100%Detected
Detection methodCoverage + fragment size shift
ConditionGene / RegionMetricPrevalenceNotes
Beckwith-Wiedemann OMIM11p15.5log2 = 0.111 in 13,700Overgrowth syndrome
Russell-Silver OMIM11p15.5 / chr7log2 = -0.281 in 100,000Growth restriction

Validation Pipeline

01

Generate

Reference-backed fragments from hg38 with condition-specific signals: coverage modifiers, variant injection, or repeat expansion at the correct genomic locus.

02

Align

BWA-MEM alignment against GRCh38 reference. All conditions achieve >95% mapping rate, confirming biological plausibility of generated sequences.

03

Detect

Category-specific detection: z-scores for aneuploidies, log2 ratios with Poisson tests for microdeletions and microduplications, VAF via pileup for monogenics and oncology hotspots, motif counting for repeat expansions.

04

Validate

GC-normalised metrics compared against detection thresholds. All 107 conditions produce the expected signal at 15% foetal fraction.

Generate Synthetic Data for Any Condition

All 107 conditions are available for synthetic cfDNA generation. Custom fetal fractions, fragment depths, and multi-condition samples supported.